- O,O-Diisopropyl S-2-phenylsulfonylaminoethyl phosphorodithioate
- Phosphorodithioic acid, O,O-bis(1-methylethyl)S-(2-((phenylsulfonyl)amino)ethyl)ester
Viscous amber liquid above 34.4 Deg c, solid below.
Selective preemergence herbicide.
Registry Numbers and Inventories.
EC Index Number
Agricultural Chemical and Pesticide
Swiss Giftliste 1
Japan ENCS (MITI)
Melting point, °C
Vapor pressure, mmHg
8E-7 (25 C)
1.25 g/cm3 (22 C)
Solubility in water
Partition coefficient, pKow
Hazards and Protection.
It is stable at 80 C for fifty hours but decomposes at 200 C in eighteen to forty hours.
Avoid bag breakage. Keep away from feed/foodstuffs. Avoid contact with sking, eyes and clothing. Wash after handling.
Proective gloves. Safey goggles.
Dusk mask if airborne dust levels are high.
Sweep us. Use good housekeeping practices. Use proper protected equipment.
Stable at 80 C for 50 hr but decomposes @ 200 C in 18-40 hr relatively stable to acids and alkalis. Decomposes slowly in light. Apparently indefinite storage life under normal ambient conditions. Decomposes on boiling bensulide is stable at 80 C for 50 hr, but decomposes at 200 C in 18 to 40 hr.
Strong oxidizing agents.
When heated to decomposition it emits very toxic fumes of nitrogen oxides, sulfur oxides ,and phosphorous oxides.
Wear a self-contained breathing apparatus in pressure-demand, MSHA/NIOSH (approved or equivalent), and full protective gear. During a fire, irritating and highly toxic gases may be generated by thermal decomposition or combustion. Use agent most appropriate to extinguish fire.
Reduced body temperature or elevated body temperature may occur. Low heart rate and abnormally low blood pressure may develop after moderate to severe poisoning. Rapid heart rate, elevated blood pressure, and changes in respiratory rate may also occur. Early giddiness, anxiety headache, and restlessness followed by ataxia, drowsiness, and confusion are common with moderate to severe exposures. Sporadic reports of human birth defects related to organophosphates have not been fully verified.
Nausea, vomiting, abdominal cramps, and diarrhea are common muscarinic effects. Both painless and frank clinical pancreatitis have been reported.
Increased bronchial secretions, bronchospasm and dyspnea occur in moderate to severe exposures. Respiratory failure and non-cardiogenic pulmonary edema may occur with severe poisonings. Acute respiratory insufficiency is the main cause of death in acute poisonings. The hydrocarbon vehicle may cause chemical pneumonitis.
Profuse sweating is common. Pallor may be noted. Dermal sensitization has been reported.
Constriction of the pupil, tearing, and blurred vision are common. Prolonged dialation of the pupils may occur. Salivation commonly occurs.
Seek medical attention. If individual is drowsy or unconscious, do not give anything by mouth; place individual on the left side with the head down. Contact a physician, medical facility, or poison control center for advice about whether to induce vomiting. If possible, do not leave individual unattended.
Move patient to fresh air. Monitor for respiratory distress. If cough or difficulty breathing develops, evaluate for respiratory tract irritation, bronchitis, or pneumonitis. Administer oxygen and assist ventilation as required. Treat bronchospasm with beta2 agonist and corticosteroid aerosols.
Systemic effects can occur from dermal exposure to organophosphates. Remove contaminated clothing, wash skin, hair and nails vigorously with repeated soap washings. Leather absorbs pesticides; all contaminated leather should be discarded. Rescue personnel and bystanders should avoid direct contact with contaminated skin, clothing, or other objects. Treatment should include recommendations listed in the oral exposure section when appropriate.
Irrigate exposed eyes with copious amounts of tepid water for at least 15 minutes. If irritation, pain, swelling, lacrimation, or photophobia persist, the patient should be seen in a health care facility. Patients symptomatic following exposure should be observed in a controlled setting until all signs and symptoms have fully resolved. Treatment should include recommendations listed in the oral exposure section when appropriate.