Cadmium mercury sulfide
- Cadmium vermilion A
- Mercury cadmium red
- C.I. Pigment Orange 23
- C.I. Pigment Red 113
wurzite lattice. Yellow to deep red as the amount of mercury increases.
Registry Numbers and Inventories.
Hazards and Protection.
Keep in a cool, dry, dark location in a tightly sealed container or cylinder. Keep away from incompatible materials, ignition sources and untrained individuals. Secure and label area. Protect containers/cylinders from physical damage.
All chemicals should be considered hazardous. Avoid direct physical contact. Use appropriate, approved safety equipment. Untrained individuals should not handle this chemical or its container. Handling should occur in a chemical fume hood.
Wear appropriate protective gloves, clothing and goggles.
Wear positive pressure self-contained breathing apparatus (SCBA).
ELIMINATE all ignition sources. Do not touch damaged containers or spilled material unless wearing appropriate protective clothing. Stop leak if you can do it without risk. Absorb or cover with dry earth, sand or other non-combustible material and transfer to containers. DO NOT GET WATER INSIDE CONTAINERS.
Extinguish using agent most appropriate for surrounding fire.
Non-combustible, substance itself does not burn but may decompose upon heating to produce corrosive and/or toxic fumes.
Contact with metals may evolve flammable hydrogen gas.
Fire may produce irritating, corrosive and/or toxic gases.
IDHL: NIOSH considers cadmium dust (as Cd) to be a potential occupational carcinogen.
O, G-A2, I-1, N-1, CP65
CNS effects of acute exposures have included delayed parkinsonism. Acute exposure - neurologic: tremor, confusion, loss of coordination, hyperreflexia, and lethargy may follow acute mercuric chloride ingestion. Chronic exposure can cause fatigue, headache, weakness, decreased concentration, anxiety, emotional lability, irritability and delirium. Smoking and industrial exposure to cadmium has resulted in increased placental levels of cadmium.
Ingestion - nausea, vomiting, abdominal pain and cramping, diarrhea, salivation, dry mouth, and substernal pain occur.
Acute inhalation of fumes can cause cough, dyspnea and chest tightness which is similar to metal fume fever, but which may progress to pneumonitis, pulmonary edema, and death due to respiratory failure in severe cases. Acute exposure can result in residual emphysema and fibrosis.
Skin eruptions and pruritus may occur.
Activated charcoal has no proven benefit in cadmium poisoning, but may be considered. Activated charcoal: administer charcoal as a slurry (240 ml water/30 g charcoal). Usual dose: 25 to 100 g in adults/adolescents. Chelation - although not demonstrably efficacious, chelation therapy may be of benefit immediately following acute exposure. Administer cana2 edta 75 mg/kg/day deep im or slow iv infusion given in 3 to 6 divided doses for up to 5 days. May be repeated for a second course after a minimum of two days drug holiday; each course should not exceed a total of 500 mg/kg body weight. Caution: bal must not be used with cadmium since the complex is nephrotoxic.
Move patient to fresh air. Monitor for respiratory distress. If cough or difficulty breathing develops, evaluate for respiratory tract irritation, bronchitis, or pneumonitis. Administer oxygen and assist ventilation as required. Treat bronchospasm with beta2 agonist and corticosteroid aerosols.
Flush skin with plenty of soap and water for at least 15 minutes while removing contaminated clothing and shoes.
Irrigate exposed eyes with copious amounts of tepid water for at least 15 minutes. If irritation, pain, swelling, lacrimation, or photophobia persist, the patient should be seen in a health care facility. Take precautions to avoid exposure of health care professionals and other individuals.
I; II; III
USCG CHRIS Code