- Phenylarsine dichloride
A clear colorless liquid.
Lacrymator poison gas, solvent for diphenylcyanoarsine.
Registry Numbers and Inventories.
Agricultural Chemical and Pesticide; Organometallic
Swiss Giftliste 1
Melting point, °C
Boiling point, °C
66 (1 torr)
Vapor pressure, mmHg
1.641 g/cm3 (13 C)
44.64 g/s2 (19 C)
1.6313 (15.3 C)
Heat of vaporization
Heat of combustion
Hazards and Protection.
Do not store in metal containers - aterial corrodes metals because of acid formed. Can be stored at ambient temperatures.
All chemicals should be considered hazardous. Avoid direct physical contact. Use appropriate, approved safety equipment. Untrained individuals should not handle this chemical or its container. Handling should occur in a chemical fume hood.
Wear appropriate clothing to prevent any reasonable probability of skin contact. Wear eye protection to prevent any possibility of eye contact.
Any self contained breathing apparatus with a full facepiece and operated in a pressure demand or other positive pressure mode or any supplied air respirator with a full facepiece and operated in a pressure demand or other positive pressure mode in combination with an auxiliary self contained breathing apparatus operated in pressure-demand or other positive pressure mode.
Keep sparks, flames, and other sources of ignition away. Keep material out of water sources and sewers. Build dikes to contain flow as necessary.
Containers may explode in heat of fire.
Incompatible with acids and bases Incompatible with oxidizing agents.
Decomposes in water.
Wear positive pressure breathing apparatus. Move container from fire area if you can do it without risk. Fight fire from maximum distance. Dike fire control water for later disposal; do not scatter the material.Extinguish with dry chemical, carbon dioxide, water spray, foam, or fog.
This material may burn but does not ignite readily.
Containers may explode in heat of fire. Fire and runoff from fire control water may produce irritating or poisonous gases. Upon decomposition hydrogen chloride and phenylarsenious oxide are emitted. Unstable. Decomposed by water.
Fire may produce irritating, corrosive and/or toxic gases.
OSHA PEL: 1910.1018 TWA 0.010 mg/m3 NIOSH REL: Ca C 0.002 mg/m3 15-minute See Appendix A NIOSH IDLH: Potential occupational carcinogen 5 mg/m3 (as As)
Abnormally low blood pressure and rapid heart rate may develop with acute arsenic poisoning. Toxic delirium and encephalopathy are complications of acute arsenic poisoning; encephalopathy may be permanent. Peripheral neuropathy leading to profound muscular weakness and wasting may be delayed by several weeks after acute exposure; recovery is usually slow or incomplete. No reproductive studies were found for dichlorophenylarsine, but arsenic compounds have been fetotoxic and teratogenic in animals. Arsenic can most likely cross the placenta. One neonatal fatality and several uneventful births have occurred in cases of acute arsenic poisoning in pregnancy. Arsenic is excreted in the breast milk. Lower doses of arsenic have not affected male fertility in animals, but toxic doses have produced effects on the testes.
Induction of vomiting is one of the bases for its use as a military agent. Gastroenteritis, sometimes with watery or bloody diarrhea, occurs with acute exposure to arsenic.
Severe respiratory irritation leading to pulmonary edema would be expected from acute exposure to dichlorophenylarsine. Respiratory failure and adult respiratory distress syndrome have occurred in acute arsenic intoxication.
Burns and blistering occur from dichlorophenylarsine. Common skin findings after either acute or chronic arsenic poisoning may include flushing, diaphoresis, palmar hyperkeratosis, peripheral edema, hyperpigmentation, brawny desquamation, and exfoliative dermatitis. Mee's lines (transverse white lines in the nails) may appear approximately five weeks after acute exposure. Shingles may also be a complication of arsenic poisoning.
Tearing and burns or strong irritation occur to the eyes. A garlic-like odor may be apparent on the breath.
Do not induce emesis - dilution: following ingestion and/or prior to gastric evacuation, immediately dilute with 4 to 8 ounces (120 to 240 ml) of milk or water (not to exceed 15 ml/kg in a child). The possible benefit of early removal of some ingested material by cautious gastric lavage must be weighed against potential complications of bleeding or perforation.
Move patient to fresh air. Monitor for respiratory distress. If cough or difficulty breathing develops, evaluate for respiratory tract irritation, bronchitis, or pneumonitis. Administer oxygen and assist ventilation as required. Treat bronchospasm with beta2 agonist and corticosteroid aerosols.
Remove all exposed clothing and jewelry taking necessary precautions to prevent secondary exposure to health care providers. Irrigate exposed areas promptly with copious amounts of water for at least 30 minutes. Wash the skin, including hair and nails, vigorously; do repeated soap washings. Discard contaminated clothing.
Irrigate exposed eyes with copious amounts of tepid water for at least 15 minutes. If irritation, pain, swelling, lacrimation, or photophobia persist, the patient should be seen in a health care facility. All patients with significant eye exposure should be carefully monitored for possible development of systemic signs and symptoms. Follow treatment recommendations in dermal exposure section where appropriate. Note: see treatment of eye exposure in the main body of this document for complete information.
I; II; III
USCG CHRIS Code
Std. Transport #